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. 2018 Jun 20;8:9403. doi: 10.1038/s41598-018-27539-2

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The double subcongenic strain SHRSPrch1.1_18.0. (a) Stroke latency under salt-loading. The Cox hazard model was used to calculate relative risks (RR) in the table below. To compare with the latency of SHRSPrch1.1 and 1_18, the data obtained in the previous study were used for these two strains (see ref.⁶). Note that the data newly obtained in this study is shown for SHRSPrch18.0 (see also ref.⁶). 1.1_18.0: SHRSPrch1.1_18.0. (b) SBP measured by telemetry during salt-loading. No significant increase was observed (n = 3). (c) Genomic construction of SHRSPrch1.1_18.0 in comparison with the original double congenic strain SHRSPrch1_18. Note that the congenic region on Chr 1 was much shorter in SHRSPrch1.1_18.0.