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. 2018 Jun 14;9:635. doi: 10.3389/fphar.2018.00635

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Copyright © 2018 Liou, Cheng, Yeh, Wu and Huang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effects of casticin (CAS) on inflammatory mediators and oxidative stress factors. (A) Western blot showing that CAS suppressed COX-2 and iNOS expression, and (B) regulated HO-1 and Nrf2 expression in lung tissue from normal (N) and OVA-stimulated (OVA) mice with or without (CAS) and prednisolone (P) treatment. (C) GSH activity and (D) MDA activity in the lung tissues of mice. The data are presented as means ± SEM of three independent experiments (n = 6). ^∗p < 0.05, ^∗∗p < 0.01 compared to OVA control mice.