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. 2018 Jun 14;9:635. doi: 10.3389/fphar.2018.00635

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Copyright © 2018 Liou, Cheng, Yeh, Wu and Huang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Casticin (CAS) inhibited THP-1 cell adherence to activated BEAS-2B cells. (A) CAS decreased the levels of ICAM-1 in BEAS-2B cells activated with TNF-α. The data are presented as means ± SEM of three independent experiments (n = 9). ^∗p < 0.05, ^∗∗p < 0.01 compared to BEAS-2B cells stimulated with TNF-α. (B) Fluorescence microscope images of THP-1 cells labeled with calcein-AM and mixed with untreated cells (normal control, N) and TNF-α-activated BEAS-2B cells in the absence or presence of CAS.