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. 2018 Jun 21;17:96. doi: 10.1186/s12943-018-0844-7

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — PTAF overexpression leads to OVCAR-3 cell migration and invasion by regulating miR-25. Wound-healing a and migration assays b showed that PTAF promoted OVCAR-3 cell migration, which was inhibited by forced expression of miR-25. n = 5 independent experiments. *P < 0.05, **P < 0.01. c A Transwell invasion assay showed that PTAF promoted OVCAR-3 cell invasion by regulating miR-25. n = 5 independent experiments. **P < 0.01. Wound-healing d and migration assays e showed that silencing of PTAF inhibited TGF-β1-driven migration in OVCAR-3 cells, which was abated by miR-25 knockdown. n = 5 independent experiments. *P < 0.05, **P < 0.01. f A Transwell invasion assay showed that knockdown of PTAF inhibited TGF-β1-induced OVCAR-3 cell invasion. n = 5 independent experiments. *P < 0.05, **P < 0.01