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. 2017 Dec 29;40(1):30–37. doi: 10.1080/0886022X.2017.1419970

Table 3.

Nature, mechanism, severity and etiology of AKI amongst participants.

Variables Total (%) Male (%) Female (%) p
Nature
 Community acquired 500 (93.30) 278 (51.90) 222 (41.40) .099
 Hospital acquired 36 (6.70) 15 (2.80) 21 (3.90)
Mechanism
 Pre renal 329 (61.40) 195 (36.40) 134 (25.00) .004
 Acute tubular necrosis 155 (28.90) 84 (15.70) 71 (13.20)  
 Glomerular 6 (1.10) 2 (0.40) 4 (0.70)  
 Interstitial 14 (2.60) 3 (0.60) 11 (2.00)  
 Vascular 10 (1.90) 3 (0.60) 7 (1.30)  
 Obstructive 22 (4.10) 6 (1.10) 16 (3.00)  
Severity AKI
 1 176 (32.80) 114 (21.30) 62 (11.50) .005
 2 128 (23.90) 65 (12.10) 63 (11.80)  
 3 232 (43.30) 114 (21.30) 118 (22.00)  
Etiologies        
 Sepsis/bacterial infection 310 (50.40) 173 (28.10) 137 (22.30)  
 Volume depletion 195 (31.60) 109 (17.70) 86 (13.90)  
 Nephrotoxins 62 (10.10) 40 (6.50) 22 (6.40)  
 Obstruction 22 (3.60) 5 (0.80) 17 (2.80)  
 Eclampsia/HELLP 11 (1.80) 0 (0.00) 11 (1.80)  
 Multiple Myeloma 5 (0.80) 3 (0.50) 2 (0.30)  
 Malignant hypertension 5 (0.80) 3 (0.30) 2 (0.20)  
 Systemic lupus 3 (0.50) 0 (0.00) 3 (0.50)  
 Lymphoma 1 (0.20) 1 (0.20) 0 (0.00)  
 Unknown 1 (0.20) 1 (0.20) 0 (0.00)  

Bold indicates the significant p values.