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. 2017 Jan 24;39(1):340–349. doi: 10.1080/0886022X.2017.1279553

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© 2017 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 3. — (A) Representative images of F4/80-stained slices of kidneys from mice of the (a) sham-operated, (b) vehicle-treated, and (c) alogliptin-treated groups (original magnification, ×200). (B) Percentage of tubulointerstitial area positive for F4/80 staining in kidneys in the sham-operated (sham, n = 10), vehicle-treated (veh, n = 8), and alogliptin-treated (alogliptin, n = 9) groups. *p < 0.001 compared with the sham-operated group. $p < 0.01 compared with the vehicle-treated group. (C) Relative expression levels of CD68 messenger RNA (mRNA) in kidneys in each group. Glyceraldehyde 3-phosphate dehydrogenase (GAPDH) mRNA was used as the internal control to adjust for unequal total mRNA content. C *p < 0.01 compared with the sham-operated group; $p < 0.05 compared with the vehicle-treated group.