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. 2018 Apr 14;164(1):339–352. doi: 10.1093/toxsci/kfy090

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© The Author(s) 2018. Published by Oxford University Press on behalf of the Society of Toxicology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs licence (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial reproduction and distribution of the work, in any medium, provided the original work is not altered or transformed in any way, and that the work is properly cited. For commercial re-use, please contactjournals.permissions@oup.com

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Figure 5. — Significant pathways altered in OH-GQDs-treated human esophageal epithelial cells. A, Clustering map of genes differentially expressed in OH-GQDs-treated and control HET-1A cells. B, Volcano plot comparing vehicle treatment versus OH-GQDs treatment. C and D, Significant pathways altered in OH-GQDs treated and control HET-1A cells. E, Quantitative real-time PCR validation for selected genes between OH-GQDs treated and control cells (n = 3). Student’s t test was performed. When compared with control cells, **p < .01, *** p < .001. (For interpretation of the references to colour in this figure legend, the reader is referred to the web version of this article.)