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. 2018 Mar 15;24:3. doi: 10.1186/s10020-018-0005-x

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© The Author(s) 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Time dependent effects of a high salt diet (HSD) on palmitate oxidation, glucose oxidation, glycolysis, and lactate oxidation in Dahl salt-sensitive rat hearts. a Palmitate oxidation, (b) Glucose oxidation, (c) Lactate oxidation, (d) ATP production, (e) Glycolysis, and (f) Proton production were measured in hearts from Dahl salt-sensitive rats fed a low salt diet, 0.3% NaCl (Control) or a HSD, 8% NaCl, for 3, 6, or 9 weeks. Energy metabolic rates were assessed in isolated working hearts. Proton production was calculated based on glycolysis and glucose oxidation rates. Contribution to ATP production was calculated from the metabolic rates assessed using the isolated working heart perfusion in Dahl salt-sensitive rats. n = 3–5 * p < 0.05 compared to Control. Values shown as mean ± SEM