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. 2018 Feb 5;19(5):413–434. doi: 10.2217/pgs-2017-0186

Table 1. . Genome-wide association study input studies for the warfarin experiment.

PMID Phenotype class Phenotype Sample size Population ancestry SNPs
18535201 Response Warfarin maintenance dose 555 European 3

19300499 Response Warfarin maintenance dose 1641 European 4

20833655 Response Warfarin maintenance dose 1952 Japanese 5

23755828 Response Warfarin maintenance dose 965 African–American 1

26265036 Response Warfarin maintenance dose 2967 Brazilian, European, Japanese, African American 16

22443383 ADEs Hemostatic factors and hematological phenotypes 951 European 9

23381943 ADEs End-stage coagulation 2100 European 23

24357727 ADEs Thrombin-generation potential phenotypes 3224 European 4

19278955 Disease/background Venous thromboembolism 4884 European 1

20212171 Disease/background C4b binding protein levels 352 European 1

20303064 Disease/background Activated partial thromboplastin time 1431 European 3

21980494 Disease/background Venous thromboembolism 2652 European 4

22216198 Disease/background Anticoagulant levels 397 European 8

22701019 Disease/background Factor XI 997 European 0

22703881 Disease/background Prothrombin time 3569 European 2

22703881 Disease/background Activated partial thromboplastin time 11851 European 9

22672568 Disease/background Venous thromboembolism 5787 European and other 5

23267103 Disease/background Coagulation factor levels 3250 European 7

23509962 Disease/background Venous thromboembolism (SNP × SNP interaction) 4291 European 37

23650146 Disease/background Venous thromboembolism 51266 European 8

25240745 Disease/background Mitochondrial DNA levels 386 European 21

25772935 Disease/background Venous thromboembolism 65734 European 10

22550155 Disease/background Platelet thrombus formation 241 European, African–American 6

A total of 23 associations from 22 genome-wide association studies (GWAS) [39–60] met the criteria for inclusion in the warfarin experiment, and their significant associations were rendered into the MVF and organized into phenotypic classes, along with 23 additional variants annotated by the Pharmacogenomics Knowledge Base (PharmGKB®) [61]. The 23 additional variants included all variants annotated by the PharmGKB as of September 2016, with a significant association with warfarin response, and were not already included in the list of variants identified from GWAS. Population descriptions are taken from the summary annotations in the original papers and the GWAS Catalog [62] , but in the PIP input files they are represented in 1000 Genomes format to replicate the detailed descriptions in the original papers as precisely as possible. Although they represent a variety of ancestries, European ancestry predominated among 17 out of 22 (77%) of the study populations.

ADE: Adverse drug event; MVF: Master variant file; PIP: Pharmacoepigenomics informatics pipeline; PMID: PubMed ID.