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. 2018 Jun 12;9:1346. doi: 10.3389/fimmu.2018.01346

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Copyright © 2018 Maertzdorf, Tönnies, Lozza, Schommer-Leitner, Mollenkopf, Bauer and Kaufmann.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Analysis of distinct pulmonary cells. (A) Gating strategy for alveolar macrophages, epithelial cells (epi), and endothelial cells (endo). (B) Heatmap displaying log2 fold differences between cells from infected and uninfected for each donor. Shown are unique transcripts from a combined list of the top 50 differentially expressed transcripts for each cell type. (C) tmod gene module enrichments in innate pulmonary cells from infected versus uninfected tissue. gd, γ/δ T cells; mait, mucosal associated invariant T (MAIT) cells; natural killer (NK) and natural killer T (NKT) cells. (D) tmod evidence plots showing ranking of individual genes from three prominently enriched gene modules in panel (C). A high resolution version of this figure is supplied as Figure S5 in Supplementary Material.