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. 2018 Apr 18;69(15):3675–3688. doi: 10.1093/jxb/ery146

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© The Author(s) 2018. Published by Oxford University Press on behalf of the Society for Experimental Biology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 3. — Loss of PLDα1 and/or PLDδ affects basal resistance against oomycetes, but not ETI mediated by RPP4. (A) Representative cotyledons showing disease phenotypes of the indicated disease classes at 7 dpi. Ten-day-old seedlings were inoculated with virulent Hyaloperonospora arabidopsidis (Hpa) isolate Noco2 or avirulent isolate Emwa1. Sporangiophores (Sp) per cotyledon were assessed at 7 dpi, and categorized into five classes as indicated by the corresponding figure keys. (B) Quantification of the number of cotyledons (n >100 for each of the indicated genotypes) per class of the indicated genotypes infected with Hpa isolate Noco2 (upper panel) or avirulent isolate Emwa1 (lower panel) based on categorization of leaf infection defined in (A). χ² test was used to test statistical significance for disease degree between Col-0 and the indicated mutant lines at 7 dpi (**P<0.01).