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. 2018 Jun 25;9:480. doi: 10.3389/fneur.2018.00480

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Copyright © 2018 Sanches, Van de Looij, Toulotte, da Silva, Romero and Sizonenko.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effect of pregnancy swimming on the pup's protein levels on the right hippocampus extracts 24 h following HI (PND4). Bars graph represent immunoblots of (A) neurons (NeuN) and migrating neurons Doublecortin (DCX), oligodendrocytes progenitors (NG2) and microglia (Iba-1); (B) cell death (ccaspase 3 and fractin) and inflammation (CD11b), (C) neurotrophins VEGF and BDNF and the TRK-B receptor and (D) astrogliosis (GFAP), glutamate receptor GLT-1 and glutamine synthetase enzyme in the four experimental groups: sedentary sham (SESH), sedentary hypoxic-ischemic (SEHI), swimming sham (SWSH) and swimming hypoxic-ischemic (SWHI). WB results are plotted normalized to SESH level expression (100%) (mean ± SEM). Significance testing was determined using one-way ANOVA followed by Duncan's post-hoc and was performed on Actin or βIII-tubulin normalized data. *HI vs. its respective SH group. ^#SWHI vs. SESH and SEHI. Significance accepted when p < 0.05.