Table 1.

Characteristics of patients with hypermagnesemia who underwent emergency hemodialysis (n = 15)

Age, years	Gender	Dose of oral MgO (mg/day)	Serum Mg before HD (mg/dL)	Serum Cre before HD (mg/dL)	Serum Cre before discharge/death (mg/dL)	eGFR before discharge/death (mL/min/1.73 m2)	NSAIDs	ARB	AKI stage	Factors related to Mg metabolism	Background factor
68	F	1980	15.1	0.54	0.26	186.4	Y	N	2		DM, HT, Parkinson's disease, pneumonia
74	M	2000	3.7	4.04	0.85	67.4	N	N	3		DM, HT, acute glomerulonephritis, pneumonia
76	F	1980	8.1	0.84	0.48	92.3	N	N	1	Vit D
78	F	2000	7.1	7.13	1.43	27.8	Y	N	3	Vit D	HT, rheumatoid arthritis,
										CKD	urinary tract infection
79	F	Dose unknown	5.3	10.29	2.95a	12.5	N	Y	3	CKD	HT, congestive heart failure, abdominal aortic aneurysm (cause of death)
82	F	1500 + Magnesium citrate 34 g + Magnesium aluminometasilicate 1.2 g	18.6	0.90	0.33	136.1	N	N	2	Ileus	HT, depression
83	F	2000	7.0	7.06	1.55	25.0	N	N	3	Hypothyroidism CKD	HT, dementia, neurogenic bladder
92	M	990	5.3	10.18	3.60	13.0	N	Y	2	CKD	HT
94	M	1980	4.5	6.00	3.28	14.4	N	Y	1	CKD	HT
59	F	1320	6.8	0.62	0.60	77.8	N	N			DM, catheter‐related bloodstream infection
69	M	1980	4.7	1.56	2.07a	26.0	N	N			DM, HT, leukemia (cause of death)
76	F	2000	6.0	9.93	8.17	4.2	N	N		CKD	DM, HT
77	M	3000	7.3	4.31	3.49	14.2	N	N		CKD	DM, HT, pneumonia
85	M	3000	5.6	5.39	5.54	8.3	N	N		CKD	DM, HT, congestive heart failure
87	F	990	6.0	5.45	4.10	8.5	N	Y		CKD	HT

Hypothyroidism and Addison's disease are risk factors of hypermagnesemia, because these diseases increase tubular magnesium (Mg) reabsorption.38.

AKI, acute kidney injury; ARB, angiotensin II receptor blocker; CKD, chronic kidney disease; Cre, creatinine; DM, diabetes mellitus; eGFR, estimated glomerular filtration rate; F, female; HD, hemodialysis; HT, hypertension; M, male; MgO, magnesium oxide; N, no; NSAIDs, non‐steroidal anti‐inflammatory drugs; Vit D, vitamin D; Y, yes

^aPatient death during admission.