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. 2018 May 10;293(26):10353–10362. doi: 10.1074/jbc.RA118.002889

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© 2018 Abou El Hassan et al.

Published under exclusive license by The American Society for Biochemistry and Molecular Biology, Inc.

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Figure 3. — IFNγ-induced STAT1 and IRF1 binding differentially correlate with histone acetylation. A, graph shows the percentages of preacetylated STAT1 and/or IRF1 sites at promoter proximal (≤5 kb) or remote (>5 kb) distances from known genes. B, graphs show the percentages of distribution of IFNγ-induced H3/4ac relative to TF binding within proximal (≤5 kb) or distal (>5 kb) regions of known gene starts. The distribution of equal numbers of randomly generated sites is also shown. C, graph shows the percentages of acetylation of different classes of STAT1- and IRF1-binding sites compared with equal numbers of randomly generated controls. Const., constitutive; Ind. or ind., induced.