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. 2018 Jul 3;9(7):751. doi: 10.1038/s41419-018-0771-y

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a, b Transwell assays revealed that u50535 knockdown repressed both migration and invasion ability in HCT116, while opposite results were found in u50535 overexpressed DLD1. c, d Wound healing test showed that u50535 knockdown would significantly slow down the ability of wound healing in HCT116, while its overexpression greatly promoted the wound healing in DLD1. Data from three independent experiments and expressed as mean ± SD. * P < 0.05; ** P < 0.01