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. 2018 May 8;16(1):83–92. doi: 10.3892/etm.2018.6140

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Copyright: © Zhou et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — The nonsense phenotype of NK cells from the peripheral blood of patients with ESCC and control subjects. (A) Representative flow cytometry images and (B) quantitative analysis of activating and inhibitory receptors and effect molecules in NK cells. (B) The column showed that the expression of p44, G2A, p46, CD158b, granzyme B, perforin, TNFα, IFNγ and Ki67. NK, natural killer; ESCC, esophageal squamous cell carcinoma; G2A, G-protein coupled receptor 132; CD, cluster of differentiation; TNF, tumor necrosis factor; IFN, interferon; SSC, side-scattered light; ns, no significance.