Skip to main content
. Author manuscript; available in PMC: 2019 Jul 1.
Published in final edited form as: Hepatology. 2018 May 10;68(1):258–273. doi: 10.1002/hep.29787

Figure 5. rhRLX protects hepatocytes from H2O2-induced cell death while upregulating GR/Bcl-2/Bcl-xL in vitro.

Figure 5

Primary mouse hepatocytes exposed to H2O2-induced oxidative stress (2mM; 5h) were pretreated with or without rhRLX (1μg/ml; 24h). (A) Cell damage was assessed by LDH, ALT and AST levels in the culture medium (n=4/group) (B) Western blot-assisted detection and relative intensity ratio of GR, Bcl-2, Bcl-xL, cleaved caspase 3. β-actin expression served as an internal control and used for normalization (n=3/group). Data shown as mean±SD. *p<0.05 vs. cells+H2O2 (Student t-test).