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. 2018 May 11;42(2):946–956. doi: 10.3892/ijmm.2018.3670

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Phosphorylation of Akt and eRK in BMSCs. HO-1 gene modification increased the phosphorylation of Akt and eRK in BMSCs in the AKI microen-vironment. Results are presented as the mean ± standard deviation (n=6). ^aP<0.05 vs. control group; ^bP<0.05 vs. BMSCs/AKI-KHS group. ERK, extracellular signal-regulated kinase; pERK, phosphorylated ERK; Akt, protein kinase B; pAkt, phosphorylated Akt; BMSCs, bone marrow-derived mesenchymal stem cells; HO-1, heme oxygenase-1; AKI, acute kidney injury; KHS, kidney homogenate supernatant; blank, blank group (BMSCs with medium); con, control group (BMSCs with normal-KHS); eGFP, enhanced green fluorescent protein.