Fig. 7.

When the implant is axially loaded immediately after implantation, a fibrin clot in the gap interface has a low modulus (~ 0.05 MPa). The loading causes axially-downward micromotion (negative z-direction) of about 93 μm (A below). In turn, this micro-motion creates strain in the interface (B, below): Principal compressive strains (and tensile strains, not shown) reach high magnitudes (> 30%) near the implant's apex, and even higher values (> 60%) near the tips of the threads (black arrows). However, in between the tips of the threads and farther away from the apex (green arrows), stains are more moderate (~ 10%) and permissible for the initial stages of bone healing, e.g., collagen formation.