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. 2018 Jun 8;16(2):2151–2160. doi: 10.3892/ol.2018.8930

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Copyright: © Tian et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — RAC1 and VASP expression levels following knockdown. (A) Expression level of VASP mRNA in VASP-shRNA MCF-7 cells, compared with NC-shRNA MCF-7 cells. *P<0.05. (B) Protein expression level of VASP protein in VASP-shRNA MCF-7 cells, compared with NC-shRNA MCF-7 cells. (C) Quantitative analysis of expression levels of VASP protein in panel (B). *P<0.05 compared with the control group. (D) Expression levels of VASP and RAC1 mRNA in RAC1-shRNA MCF-7 cells, compared with NC-shRNA MCF-7 cells. *P<0.05. (E) Protein expression level of VASP and RAC1 protein in RAC1-shRNA MCF-7 cells, compared with NC-shRNA MCF-7 cells. (F) Quantitative analysis of expression levels of VASP and RAC1 protein in panel (E). *P<0.05 compared with the control group. RAC1, Ras-related C3 botulinum toxin substrate 1; VASP, vasodilator-stimulated phosphoprotein; NC, negative control; shRNA, short hairpin RNA.