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. 2018 Jul 10;9(7):773. doi: 10.1038/s41419-018-0806-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Normal fibroblasts (HFF) (a, e) and representative cell lines from the “highly responding group” MDA-MB-231 (b, f), SF539 (c, g), and Colo357 (d, h) harboring doxycycline-inducible PSMD1 shRNA were either doxycycline treated to induce PSMD1 shRNA expression or left untreated. The levels of UPR markers ATF4 and p-eIFα were analyzed by immunoblot, and of CHOP and the spliced XBP-1 (sXBP-1) mRNA were measured by qPCR. *p < 0.05, **p < 0.01