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. 2018 Mar 2;15(4):302–313. doi: 10.1177/1479164118759220

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© The Author(s) 2018

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — In vivo inhibition of NFAT modifies the expression of oxidative stress and inflammatory markers in the aorta of young IGF-II/LDLR^–/–ApoB^100/100 mice. Gene expression of oxidative stress targets (a) NOX4, (b) NOX2, (c) GLO1 and (d) catalase, as well as inflammatory targets (e) CD68, (f) IL-6 and (g) OPN were quantified by qPCR in the aortas of young IGF-II/LDLR^–/–ApoB^100/100 mice from Study I and II. HPRT and β-actin were used as endogenous controls and data were expressed relative to each control group. N = 5 and 7 for control and treated males, respectively; N = 8 for female groups.

*p < 0.05 and **p < 0.01.