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. 2018 Jul 11;37:144. doi: 10.1186/s13046-018-0808-1

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — STK25 modulates glycolysis via GOLPH3. Culture media from controls and GOLPH3-overexpressing or GOLPH3-depleted CRC cells were analyzed to measure relative glucose uptake (a), lactate production (b), and lactate:glucose ratio (c). Knockdown of GOLPH3 decreases the promotion of glucose uptake (d) and lactate production (e) induced by STK25 depletion in LoVo cells. Data are expressed as mean ± SD. *, P < 0.05