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. 2018 Jul 10;11:3979–3987. doi: 10.2147/OTT.S158316

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© 2018 Zhang et al. This work is published and licensed by Dove Medical Press Limited

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Overexpression of CDR1as antagonized the availability of miR-7 for tumor growth.

Notes: (A) The expression of CDR1as in normal lung epithelial cells HBE and lung cancer cell lines A549 and H460 were determined by qPCR. (B and C) The efficiency of knockdown or overexpression in A549 and H460 was determined. (D) The cell vitalities of two cell lines were estimated by CCK-8 assay. (E and F) The apoptosis and cell cycle were analyzed by flow cytometry. *P<0.05, **P<0.01, ***P<0.001, data represent the mean ± SD. Control bars are missing from part C as the level of control group was too low.

Abbreviations: qPCR, quantitative polymerase chain reaction; NC, negative control.