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. 2018 Jul 10;9:851. doi: 10.3389/fphys.2018.00851

Table 2.

Reported localization and proposed physiological and pathophysiological relevance of liver aquaglyceroporins.

Aquaglyceroporins Cell type Subcellular location Suggested physiological role Suggested pathophysiological involvement
AQP3 Kupffer cells (h) Undefined Repopulation of Kupffer cells in liver regeneration Kupffer cell migration and proinflammatory cytokine secretion
Hepatocytes (h) Undefined Uptake of glycerol? Insulin resistance?
Stellate cells (h) Undefined Cell activation Hepatic steatosis (I148M variant)
AQP7 Hepatocytes (h, m) Cyt; others? TAG synthesis; uptake of glycerol? Insulin resistance
Cholangiocytes (h) APM Unknown
Endothelial cells (h) APM Unknown
AQP9 Hepatocytes (h, m, r) BLPM Uptake of portal glycerol (gluconeogenesis; TAG synthesis); import of water from portal blood (bile formation); urea extrusion; arsenic detoxification; liver regeneration NAFLD; NASH; obesity; T2D; hepatocellular carcinoma
Cholangiocytes (h) Undefined Unknown
AQP10 Hepatocytes (h) Undefined Exit of glycerol during lipolysis

APM, apical plasma membrane; BLPM, basolateral plasma membrane; h, human; m, mouse; NAFLD, non-alcoholic fatty liver disease; NASH, non-alcoholic steatohepatitis; r, rat; TAG, triacylglycerols; T2D, type 2 diabetes mellitus.