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Electroacupuncture modulated the apoptosis factors of Bcl-2 and Bax in injured rats. At translational levels, EA treatment profoundly inhibited the model construction-mediated down-regulation of Bcl-2 and Bax and the Bcl-2/Bax ratio (data are expressed as the percentage of the value of sham group) (*, vs. MCAO group; * p<0.05/*** p<0.001; #, vs. sham group; ## p<0.01/### p<0.001) (n=4 in each group).
