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. 2018 Jul 18;18:85. doi: 10.1186/s12871-018-0554-0

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — a. Latency to anesthesia induction measured by the time until immobility in WAG/Rij rats. In KE and KS groups the latency to anesthesia was significantly longer (p < 0.0001, p = 0.02, respectively) compared to control (standard diet, SD); b. Blood βHB level was significantly elevated in KE and KS groups, compared to control (p < 0.0001, p < 0.0001, respectively) and compared to their baseline (p < 0.0001, p < 0.0001, respectively; interaction: F_2,21 = 51.23, p < 0.0001; time: F_1,21 = 151, p < 0.0001; treatment: F_2,21 = 37.44, p < 0.0001). Bar on left represents baseline value, bar on the right represents value after treatment in each group; c. There was a positive correlation between latency to anesthesia induction and blood βHB levels when all data point was considered (R² = 0.4862); d. There was a strong positive correlation between latency to anesthesia induction and blood βHB levels when the group means were considered (R² = 0.995)