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. 2018 Jul 20;9:2873. doi: 10.1038/s41467-018-05357-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Alterations of sulfur and cysteine/methionine metabolism pathways in the MLI microbiome of pediatric IBD patients. a Taxon-specific sulfur or cysteine/methionine metabolism-related functions that were significantly changed in IBD. Heatmap shows the average LFQ intensity for each subgroup of samples. Each row represents one protein group, and the corresponding KO id, KO name and taxonomic assignment were indicated in the right panel. b Pathways relating to H₂S production, l-cysteine biosynthesis, and degradation as represented by the identified differentially abundant proteins. Pathway was adapted from KEGG pathways (map00270 and map00920, version 5/8/2017)