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. 2018 Jul 21;15:211. doi: 10.1186/s12974-018-1245-y

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — In situ hybridization of miR-155 in the rat TLE model. a–c A weak miR-155 expression was seen in neurons of the control rats in the DG, CA1, and entorhinal cortex (EC). d–l Increased expression compared to control was observed in both neurons and glia of the rats at the acute, latent, and chronic stages. d Co-localization was found with neurons (NeuN; d, inset a), microglia (IBA-1; d, inset b), and astrocytes (GFAP; e, inset c). Additionally, miR-155 expression was seen in blood vessels (e, inset d). Scale bar 100 μm; arrowheads indicate cells with glial morphology