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. 2018 Jul 17;9:250. doi: 10.3389/fgene.2018.00250

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Copyright © 2018 Divate and Cheung.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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BYL719 induces differentially enriched ATAC-seq peaks in T47D cells. (A) A volcano plot showing the differentially enriched peaks between BLY719 and DMSO-treated T47D cells. In total, 29,750 regions were found with differentially enriched ATAC-seq signals. Out of which 18,683 regions were gained-open (increased in chromatin accessibility) and 11,067 were gained-closed regions (decreased in chromatin accessibility) before and after BLY719 treatment of T47D cells. (B) Principal component analysis plot for the control and treatment replicates.