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. 2018 Jul 17;11:248. doi: 10.3389/fnmol.2018.00248

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Copyright © 2018 Sun, Zhou, Ba, Feng, Hu, Cheng, Liu, Guo, Xiao, Jiang, Xiong, Hao, Chen and Jiang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effect of OT on capsaicin-induced facilitation of spontaneous excitatory transmission in Lamina II neurons. (A) Effect of OT (1 μM) on glutamatergic spontaneous excitatory transmission in Lamina II neurons. Lower panel is the emphasized current traces as the dash line indicated. (B,C) Analyzed data of the frequency (B) and amplitude (C) of spontaneous EPSC after OT application in Lamina II neurons. Mean ± SEM, n = 6. Paired Student’s t-test. (D) Effect of capsaicin (1 μM) on glutamatergic spontaneous excitatory transmission in Lamina II neurons. Lower panel is the emphasized current traces as the dash line indicated. (E) Effect of OT (1 μM) on capsaicin-induced glutamatergic spontaneous excitatory transmission in Lamina II neurons. Lower panel is the emphasized current traces as the dash line indicated. (F,G) Analysis of the frequency (F) and amplitude (G) of spontaneous EPSC after OT co-application with or without capsaicin in Lamina II neurons. Mean ± SEM, n = 11; ^∗P < 0.05, ^∗∗P < 0.01. One-way ANOVA followed by two-tailed t-test with Bonferroni correction.