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. 2018 Jul 24;7:F1000 Faculty Rev-1127. [Version 1] doi: 10.12688/f1000research.14136.1

Figure 1. Effect of PPARγ activation on various tissues.

Figure 1.

ACSL1, acyl-CoA synthetase long chain family member 1; CD36, cluster of differentiation 36; CPT1b, carnitine palmitoyltransferase 1B; FA, fatty acid; FFA, free fatty acid; FGF, fibroblast growth factor; GK, glucokinase; GLUT, glucose transporter; GlyK, glycerol kinase; IRS2, insulin receptor substrate 1; LPL, lipoprotein lipase; NF-κB, nuclear factor kappa-light-chain-enhancer of activated B cells; MCP-1, monocyte chemoattractant protein 1; MMP-9, matrix metalloproteinase 9; NO, nitric oxide; PAI-1, plasminogen activator inhibitor type 1; PCK2, peroxisome proliferator-activated receptor gamma 2 binding site; PEPCK, phosphoenolpyruvate carboxykinase; PI3K, phosphoinositide 3-kinase; PPARγ, peroxisome proliferator-activated receptor γ; PPRE, peroxisome proliferator-activated receptor response element; RXR, retinoid X receptor; STAT6, signal transducer and activator of transcription 6; TG, triglyceride; TNF-α, tumor necrosis factor α; TZD, thiazolidinedione; VCAM-1, vascular cell adhesion molecule-1; VEGF, vascular endothelial growth factor.