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. 2018 Jul 2;10(7):1823–1836. doi: 10.1093/gbe/evy141

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© The Author(s) 2018. Published by Oxford University Press on behalf of the Society for Molecular Biology and Evolution.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Fig. 1. — —(A) Symptoms induced by the ancestral and evolved TEV genotypes. Upper panel: from left to right, (a) healthy plant, (b) chlorotic spots observed in a plant of passage 2 (P2) infected with one of the TEV-AS13 lineage, (c) the same for a plant infected with a one of the TEV-AS13/2b lineages, and (d) a plant from the third passage (P3) infected with lineage L4 of TEV-FINK. White arrows indicate the location of some chlorotic spots. Lower panel: (e) and (f) plants of passage P3 infected with lineage L1 of TEV-FINK showing the typical etching of TEV, (g) comparison of plants from the fifth passage (P5): healthy (left) or infected with lineage L1 of TEV-FINK (right), thus displaying no symptoms or the typical etch mosaic of TEV, respectively. (B) Evolution of the first principal component (PC1) summarizing the six pathogenicity-related traits. See text for a detailed description of the contribution of the different traits into PC1. Different symbols correspond to the different independently evolved lineages.