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. 2018 May 7;18(1):254–260. doi: 10.3892/mmr.2018.8984

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Histopathological analysis of femoral head cartilage by hematoxylin and eosin staining. Necrotic trabeculae (black arrows) or newly formed trabeculare (long white arrows), necrotic bone marrow and fibrous tissue (short white arrows) was observed in (A) inherited ANFH, (B) subject II-1 and (C) sporadic ANFH. Empty lacunae without osteocytes (black triangles) were observed in both subject II-1 and patients with sporadic ANFH. Subject II-1 was a family member with ANFH that did not possess the c.3508G>A mutation in collagen type II α1 chain that other affected family members had. No observable differences were identified between inherited and sporadic ANFH. ANFH, avascular necrosis of the femoral head. (Light microscope, magnification ×40).