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. 2018 May 9;18(1):349–357. doi: 10.3892/mmr.2018.8998

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Copyright: © Cui et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 8. — The model applied in the present study. Red arrows indicate the effect of Bap, and green arrows indicate the effect of ANXA1. Bap, benzo[a]pyrene; OS, oxidative stress; ANXA1, Annexin A1; PTEN, phosphatase and tensin homolog; FAK, focal adhesion kinase; PI3K, phosphatidylinositol 3-kinase; Akt, protein kinase B; p-, phosphorylated; SOD, superoxide dismutase; GPX, glutathione peroxidases; MDA, malondialdehyde; LDH, lactic dehydrogenase; ROS, reactive oxygen species; Bcl-2, B-cell lymphoma 2; Bax, Bcl-2-associated X protein.