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. 2018 May 23;18(1):779–788. doi: 10.3892/mmr.2018.9058

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Copyright: © Qu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Proliferation, migration and invasion of SAOS2 cells are enhanced following transfection with WWOX-AS1-si (magnification, ×200). (A) Proliferation of SAOS2 cells transfected with WWOX-AS1-si was significantly increased compared with the NC group. (B) Scratch wound-healing assays revealed that migration of SAOS2 cells transfected with WWOX-AS1-si was significantly enhanced compared with the NC group. (C) Matrigel invasion assays revealed that invasion of SAOS2 cells transfected with WWOX-AS1-si was significantly enhanced compared with the NC-si group. *P<0.05 vs. the NC-si group. WWOX, WW domain-containing oxidoreductase; WWOX-AS1, WWOX antisense RNA 1; NC, negative control; si, small interfering RNA.

Figure 5. — Proliferation, migration and invasion of SAOS2 cells are enhanced following transfection with WWOX-AS1-si (magnification, ×200). (A) Proliferation of SAOS2 cells transfected with WWOX-AS1-si was significantly increased compared with the NC group. (B) Scratch wound-healing assays revealed that migration of SAOS2 cells transfected with WWOX-AS1-si was significantly enhanced compared with the NC group. (C) Matrigel invasion assays revealed that invasion of SAOS2 cells transfected with WWOX-AS1-si was significantly enhanced compared with the NC-si group. *P<0.05 vs. the NC-si group. WWOX, WW domain-containing oxidoreductase; WWOX-AS1, WWOX antisense RNA 1; NC, negative control; si, small interfering RNA.