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. 2018 Jul 25;9:2908. doi: 10.1038/s41467-018-05374-3

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — MosGILT negatively impacts the transmission of P. berghei sporozoites. a Liver Plasmodium burden 40 hours after intradermal injection of 2500 P. berghei sporozoites incubated with either rGILT or BSA (100 µg/ml each). Data pooled from five independent experiments (mean ± SD, n = 29 BSA, n = 28 rGILT, Welch’s t-test, *p < 0.05). b Liver Plasmodium burden 40 hours after intravenous injection of 2000 rGILT or BSA-treated sporozoites (100 µg/ml each). Data are pooled from two independent experiments (mean ± SD, n = 14 BSA, n = 16 rGILT, Welch’s t-test, *p < 0.05). c Mice actively immunized with either rGILT or ovalbumin (OVA) were each bitten by three P. berghei-infected A. gambiae mosquitoes. The liver Plasmodium burden was analyzed 40 hours after the infectious bites. Data are pooled from three independent experiments (mean ± SD, n = 29 Ova, n = 29 rGILT, Welch’s t-test, *p < 0.05)