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. 2018 Jul 27;8:11304. doi: 10.1038/s41598-018-29649-3

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Immunocytochemistry of fibronectin, COL1A1, F-actin and αSMA in HCFs. The left panels show cells that were stained with DAPI. The middle panels show cells stained for either COL1A1, fibronectin, F-actin orαSMA. The right panels show merged image. The expression of COL1A1 and fibronectin is shown in the upper row, αSMA in the middle row, and phalloidin in the lower row. These components were upregulated in HCFs with ATX treatment, and the fibrotic responses were significantly suppressed by HA130 treatment. Bar, 200 μm.