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Endogenous estrogens allow flow‐mediated outward remodeling of resistance arteries in estrogen receptor α mutated at cysteine 451 (ERα‐C451A) mice but not in ERα‐AF20 mice. A, Arterial diameter was measured in response to stepwise increases in pressure in mesenteric arteries submitted chronically to high flow (HF) or to normal flow (NF). Arteries were isolated from wild‐type (n=12; B), ERα−/− (n=8; C), ERα‐C451A (n=5; D), or ERα‐AF20 (n=7; E) mice. NS indicates not significant.
