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. 2018 Jul 31;8:11470. doi: 10.1038/s41598-018-29929-y

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(A) Eight days after Wistar rats were intracranially injected with C6 cells, creation of the animal models was confirmed by MRI. (B) Survival curves for the animals in the four experimental groups. (C) Eighteen days after intracranial injection of C6 cells, intracranial tumor growth was significantly inhibited in the combination-treated group. (D) Immunohistochemistry analysis of p-AKT(Ser473) and p-4EBP1(Thr37/46) in the implanted tumors. (E) Twenty-four normal rats were intravenously injected with saline, 2 × 10⁷ pfu NDV, or 5 × 10⁷ pfu NDV once every 3 days for a total of three injections, and blood was then collected for routine and biochemical examination. White blood cell and platelet counts differed significantly among the groups, but all values were within the normal ranges. The remaining indices did not differ significantly among the groups (*P < 0.05, n = 6).