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. 2018 Jul 25;9:1713. doi: 10.3389/fimmu.2018.01713

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Copyright © 2018 Huang, He, Liang, Hu, Jiang, Yang, Mei, Zhu, Yu, Kijlstra, Yang and Hou.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Aryl hydrocarbon receptor (AhR) knockout leads to blood–retinal barrier (BRB) breakdown in experimental autoimmune uveitis (EAU) mice. (A) Representative Evans blue images of retinas in AhR^−/− and AhR^+/+ non-immunized or EAU mice. Scale bar, 30 µm (n = 4/group). (B) Left, representative Western blotting images of claudin-5, occludin and zonula occludens-1 (ZO-1) in retinas of AhR^−/− and AhR^+/+ EAU mice. Right, quantification of relative fold change of claudin-5, occluding, and ZO-1 expressions (n = 4/group; mean ± SD; **p < 0.01; ***p < 0.001; unpaired Student’s t-test).