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. 2018 May 29;18(2):1582–1590. doi: 10.3892/mmr.2018.9084

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Copyright: © Xu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — Representative immunoblots and quantified results of (A) Bcl-2 and Bax, (B) p-PI3K and PI3K, (C) p-AKT and AKT proteins by western blot analysis 28 days after myocardial infarction. The data are expressed as the mean ± standard deviation. *P<0.05 vs. sham, ^#P<0.05 vs. NS. n=6 in each group. Representative immunoblots and quantified results of (D) p-GSK3β and GSK3β and (E) Caspase3 and Cleaved-caspase3 proteins by western blot analysis 28 days after myocardial infarction. The data are expressed as the mean ± standard deviation. *P<0.05 vs. sham, ^#P<0.05 vs. NS. n=6 in each group. PI3K, phosphoinositide 3-kinase; GSK3β, glycogen synthase kinase-3β.

Figure 6. — Representative immunoblots and quantified results of (A) Bcl-2 and Bax, (B) p-PI3K and PI3K, (C) p-AKT and AKT proteins by western blot analysis 28 days after myocardial infarction. The data are expressed as the mean ± standard deviation. *P<0.05 vs. sham, ^#P<0.05 vs. NS. n=6 in each group. Representative immunoblots and quantified results of (D) p-GSK3β and GSK3β and (E) Caspase3 and Cleaved-caspase3 proteins by western blot analysis 28 days after myocardial infarction. The data are expressed as the mean ± standard deviation. *P<0.05 vs. sham, ^#P<0.05 vs. NS. n=6 in each group. PI3K, phosphoinositide 3-kinase; GSK3β, glycogen synthase kinase-3β.