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. 2018 Jun 25;40(3):1203–1222. doi: 10.3892/or.2018.6520

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Copyright: © Teng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Ratios of proteins involved in the mitogen-activated protein kinase pathway between AR-resistant and non-resistant gastric cancer cells. Ratios of (A) p-DUSP1/total DUSP1 (B) p-JNK/total JNK, (C) p-ERK/total ERK and (D) p-P38/total P38 in MGC803 and MGC803-AR cells, respectively. Ratios of (E) p-DUSP1/total DUSP1 (F) p-JNK/total JNK, (G) p-ERK/total ERK and (H) p-P38/total P38 in MKN45 and MKN45-AR cells, respectively (n=3; Student's t-test; *P<0.05, **P<0.01, ***P<0.001). AR, apatinib-resistant; p-/phospho, phosphorylated DUSP, dual-specificity phosphatase-1; JNK, c-Jun N-terminal kinase; ERK, extracellular signal-regulated kinase; p-, phosphorylated; PARP, poly(ADP-ribose) polymerase.