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Schematic description of the EET-PGC-1α-HO-1 interplay, increased mitochondrial biogenesis and function. The EET-A-mediated activation of PGC-1α and HO-1 leads to increased oxygen consumption, increased mitochondrial fusion, reduced visceral adipose tissue deposition that together with increased insulin sensitivity contributes to reduced sodium retention. These factors in concert lead to a lowering of blood pressure and an improvement in renal and cardiovascular function.
