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. 2018 Jul 30;11:263. doi: 10.3389/fnmol.2018.00263

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Copyright © 2018 Yang, Seo, Oh, Ryu, Kim, Lee, Ryu and Choe.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Regulation of locomotor activity by PKG binding to GluA1-CT domain in the NAc after repeated cocaine administration. (A) Timeline for locomotor activity analysis. (B) Intra-NAc infusion of Tat-GluA1-i, but not Tat-GluA1-c, significantly attenuated the repeated cocaine-induced increase in locomotor activity (Treatment, F_(5,54) = 68.82; df = 59; n = 10 rats per group; p < 0.05; *p < 0.05 vs. vehicle + repeated saline group; ^#p < 0.05 vs. vehicle + repeated cocaine group). One-way ANOVA followed by Tukey’s multiple comparisons test.