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. 2018 Aug 2;12:225. doi: 10.3389/fncel.2018.00225

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Copyright © 2018 Lu, Liu, Mai, Zang, Shen, Zhang and Xu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Rosuvastatin protected against HT via inhibiting the mitogen-activated protein kinase (MAPK) pathway. Western blot analysis of p-c-Jun, phosphorylated c-Jun-N-terminal kinase (p-JNK) and p-p38 expression levels at 24 h after reperfusion (n = 6 in each group); the results are normalized to β-actin. The values are expressed as the mean ± SEM. *P < 0.05, **P < 0.01 and ***P < 0.001 vs. the SHAM group, ^#P < 0.05 and ^###P < 0.001 vs. the HT group, ^&&&P < 0.05 vs. the normal-dose rosuvastatin-treated group.