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. 2018 Jul 25;2018:3685391. doi: 10.1155/2018/3685391

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Copyright © 2018 Meifang Zhu et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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The effect of gastrodin (GAS) on the expression of 14-3-3η in H9c2 cells exposed to anoxia/reoxygenation (A/R) injury. 14-3-3η expression was evaluated by Western blot, and β-actin was used as an internal control. (a) The expression of the 14-3-3η protein significantly increased in the A/R group (^∗∗P < 0.01 versus control group). In the concentration-effect GAS + A/R group, the expression of 14-3-3η in the GAS + A/R group was higher than that in the A/R group (^#P < 0.05 and ^##P < 0.01 versus A/R group), and the 20 mg/L GAS + A/R group presented with the highest expression of 14-3-3η (^&&P < 0.01 versus 5, 10, 40, 80, 160, and 320 mg/L GAS + A/R group). (b) The expression of the 14-3-3η protein significantly increased in the A/R group (^∗∗P < 0.01 versus control group). And the expression of the 14-3-3η protein significantly increased in both the GAS + A/R group and the NC + GAS + A/R group (^##P < 0.01 versus A/R group) but decreased in the siRNA14-3-3η + GAS + A/R group (^▲▲P < 0.01 versus GAS + A/R group). Data are expressed as the mean ± SEM (n = 3).