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. 2018 Aug 8;4(2):dvy021. doi: 10.1093/eep/dvy021

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© The Author(s) 2018. Published by Oxford University Press.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 3: — maternal PRS- and FLX-induced changes in Adar, Adarb1 and Htr2c gene expression in the offspring brain. (A) Fold changes in mRNA gene expression of Adar (A₁), Adarb1 (A₂) and Htr2c (A₃) in the PFC of offspring of PRS (O-PRS) and control (O-C) dams treated with VEH or FLX prior to gestation. (B) Fold changes in mRNA gene expression of Adar (B₁), Adarb1 (B₂) and Htr2c (B₃) in the amygdala of offspring of PRS (O-PRS) and control (O-C) dams treated with VEH or FLX prior to gestation. Fold change values are presented as means ± SE of the fold change relative to O-C). #P < 0.075. *P < 0.05. **P < 0.001. Gray panels show previously published data [22]. N's, Adar, PFC: VEH, O-C 5, O-PRS 8, FLX, 6,7; amygdala: VEH, 4,9, FLX 7,5. Adarb1, PFC: VEH, O-C 5, O-PRS 8, FLX, 7,7; amygdala: VEH, 4,8, FLX 7,6. Htr2c, PFC: VEH, O-C 6, O-PRS 8, FLX, 7,8; amygdala: VEH, 4,8, FLX 6,6.