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. 2018 Mar 29;125(1):86–96. doi: 10.1152/japplphysiol.01138.2017

Fig. 1.

Fig. 1.

Skeletal muscle citrate synthase activity, left ventricle (LV) brain natriuretic peptide (BNP) mRNA levels, body weight, and cardiac remodeling in nonsham sedentary control (CON), aortic-banded heart failure sedentary (HF), and aortic-banded heart failure interval exercise-trained (HF-IT) animals. A: exercise training prevents decreased skeletal muscle citrate synthase activity observed in HF animals (1-way ANOVA, P < 0.05). B: exercise training attenuates increased BNP mRNA levels in the LV of the HF group (1-way ANOVA, P < 0.05). C: aortic banding generates cardiac hypertrophy evident by increased postmortem LV + septum (LV+S), right ventricle (RV), and left + right atria (ATRIA) weight that is not inhibited by exercise training (1-way ANOVA, P < 0.05). Body weight was the same in all groups. *Post hoc vs. CON (P < 0.05); †post hoc vs. HF-IT (P < 0.05); #post hoc vs. CON (P < 0.10). n = 7, 7, and 7 for CON, HF, and HF-IT, respectively.