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. 2018 Aug 11;15:224. doi: 10.1186/s12974-018-1269-3

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — K284-6111 on neuroinflammatory and amyloidogenic responses in microglial BV-2 cells and astrocytes. NO level was measured in K284-6111-treated microglial BV-2 cells (a) and astrocytes (b). CHI3L1, iNOS, COX-2, and Iba-1 proteins were detected by Western blotting using specific antibodies in K284-6111-treated microglial BV-2 cells (c). CHI3L1, iNOS, COX-2, and GFAP proteins were detected by Western blotting using specific antibodies in K284-6111-treated cultured astrocytes (d). APP and BACE1 proteins were detected by Western blotting using specific antibodies in K284-6111-treated microglial BV-2 cells (e) and cultured astrocytes (f). Each value under the band is mean ± S.D. from three experiments (c, d, e, f). ^#Significantly different from control group (p < 0.05). *Significantly different from LPS-treated group (p < 0.05)